Published in AIDS Weekly, June 19th, 1995
Researcher Roberto Paganelli and colleagues found that the immunologic mechanisms in this condition seem to differ from those known in primary hyper-IgE syndrome because CD4 T H2 type cells, which are currently believed to have a role in IgE production, are severely depleted in HIV-positive patients ("Immunologic Aspects of Hyperimmunoglobulinemia E-Like Syndrome in Patients with AIDS," Journal of Allergy and Clinical Immunology, May 1995;5(1):995-1002).
Hypereosinophilia accompanied by...
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