Temporomandibular Joint Dysfunction Syndrome

New temporomandibular joint dysfunction syndrome research from University of Minnesota discussed

Published in Biotech Business Week, January 12th, 2009

"The influence of analgesic agents on neurons activated by stimulation of the temporomandibular joint (TMJ) region is not well defined. The spinomedullary junction [trigeminal subnucleus caudalis (Vc)/C1-2] is a major site of termination for TMJ sensory afferents," investigators in the United States report.

"To determine whether estrogen status influences opioid-induced modulation of TMJ units, the classical opioid analgesic, morphine, was given to ovariectomized (OvX) rats and OvX rats treated for 2 days with low-dose (LE2) or high-dose (HE2) 17 beta-estradiol-3-benzoate. Under thiopental anesthesia, TMJ units in superficial and deep laminae at the Vc/C1-2 junction...

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Source: Biotech Business Week (2009-01-12)

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