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Albert Einstein College of Medicine, New York
GluR2-lacking AMPA receptors linked to death of hippocampal CA1 pyramidal neurons
November 4th, 2005
GluR2-lacking AMPA receptors have been identified as a key therapeutic target in acute ischemic stroke. "Transient global or forebrain ischemia induced experimentally in animals can cause selective, delayed neuronal death of hippocampal CA1 pyramidal neurons," neuroscientists in New York. "A striking feature is a delayed rise in intracellular free Zn2+ in CA1 neurons just before the onset of histologically detectable cell death." In a recent study, K.M. Noh and coauthors at the Albert Einstein College of Medicine found that "alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)-type glutamate receptors (AMPARs) at Schaffer...
Source: Biotech Law Weekly (2005-11-04)
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