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Boston University
Neutrophil respiratory burst response to influenza A virus is differentially modulated
November 4th, 2005
According to a study from the United States, respiratory innate immune proteins differentially modulate the neutrophil respiratory burst response to influenza A virus (IAV). "Oxidants and neutrophils contribute to lung injury during IAV infection. Surfactant protein (SP)-D plays a pivotal role in restricting IAV replication and inflammation in the first several days after infection," wrote M.R. White and colleagues, Boston University. "Despite its potent anti-inflammatory effects in vivo, preincubation of IAV with SP-D in vitro strongly increases neutrophil respiratory burst responses to the virus. Several factors are shown to modify this apparent...
Source: Biotech Law Weekly (2005-11-04)
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