University of Kentucky
4-hydroxynonenal may result in increased levels of intracellular zinc
Published in Biotech Law Weekly, March 24th, 2006
According to recent research from the United States, "Accumulating evidence implicates oxidative stress in the pathogenesis of several neurodegenerative disorders including Alzheimer's disease (AD). Increased lipid peroxidation, decreased levels of polyunsaturated fatty acids, and increased levels of 4-hydroxynonenal (HNE) have been demonstrated in AD brain."
J.L. Smith and colleagues from the University Kentucky explained, "Proteins responsible for zinc export are localized on the plasma membrane and may be vulnerable to damage from lipid peroxidation.
"To...
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Source: Biotech Law Weekly (2006-03-24)
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