Published in Blood Weekly, November 15th, 2001
F. Galietti and colleagues at the University of Turin's San Luigi Gonzaga Hospital in Orbassano conducted a study to "evaluate the expression of such proteins in peripheral blood human monocyte cultures infected with strains of different mycobacterial pathogens."
Unlike less-pathogenic mycobacteria, M. tuberculosis induces a significant upregulation of p53 and p21 in blood cells, they found.
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