Acute Myeloid Leukemia

Mutation may contribute to AML

Published in Blood Weekly, August 28th, 2003

Researchers at Washington University School of Medicine in St. Louis have found that a chromosome defect often seen in human acute myeloid leukemia (AML) can cause the same disease in mice when combined with a genetic defect in a molecule known as a tyrosine kinase receptor.

The study is published online and in the August 5, 2003, issue of the Proceedings of the National Academy of Sciences USA.

"The findings indicate that tyrosine kinases may be an important class of cancer-causing genes in leukemia," said principal investigator Michael H. Tomasson, MD, assistant professor of genetics and of medicine. "It further suggests that drugs designed...

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Source: Blood Weekly (2003-08-28)

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