Published in Blood Weekly, December 2nd, 2004
According to a study from England, "Nitric oxide (NO) is thought to protect against the damaging effects of myocardial ischemia-reperfusion injury, whereas xanthine oxidoreductase (XOR) normally causes damage through the generation of reactive oxygen species. In the heart, inorganic nitrite (NO2 ) has the potential to act as an endogenous store of NO liberated specifically during ischemia."
A. Ahluwalia of Queen Mary's School of Medicine and Dentistry explained, "Using a detection method that we developed, we report that under...
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