Ischemia Genetics
University of Pittsburgh, Department of Surgery reports research in ischemia genetics
Published in Blood Weekly, February 15th, 2007
A new study, "Increasing numbers of hepatic dendritic cells promote HMGB1-mediated ischemia-reperfusion injury," is now available. "Endogenous ligands released from damaged cells, so-called damage-associated molecular pattern molecules (DAMPs), activate innate signaling pathways including the TLRs. We have shown that hepatic, warm ischemia and reperfusion (I/R) injury, generating local, noninfectious DAMPs, promotes inflammation, which is largely TLR4-dependent," scientists in the United States report.
"Here, we demonstrate that increasing dendritic cell (DC) numbers enhance inflammation and organ injury after hepatic I/R. High-mobility group box 1 (HMGB1), a NF...
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Source: Blood Weekly (2007-02-15)
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