Physiology
Studies conducted at Ohio State University, Davis Heart and Lung Research Institute on physiology recently published
Published in Blood Weekly, April 5th, 2007
A report, "Endothelial nitric oxide synthase (NOS3) knockout decreases NOS2 induction, limiting hyperoxygenation and conferring protection in the postischemic heart," is newly published data in American Journal of Physiology - Heart and Circulatory Physiology. "Although it has been shown that endothelial nitric oxide synthase (eNOS)-derived nitric oxide downregulates mitochondrial oxygen consumption during early reperfusion, its effects on inducible NOS (iNOS) induction and myocardial injury during late reperfusion are unknown. Wild-type (WT) and eNOS(-/-) mice were subjected to 30 min of coronary ligation followed by reperfusion," scientists in the United States report.
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Source: Blood Weekly (2007-04-05)
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