Ischemia Cell Biology
New ischemia cell biology findings from University Hospital published
Published in Blood Weekly, May 17th, 2007
A new study, "A peptide inhibitor of c-Jun NH2-terminal kinase reduces myocardial ischemia-reperfusion injury and infarct size in vivo," is now available. "The c-Jun NH(2)-terminal kinase (JNK) pathway of the mitogen-activated protein kinase (MAPK) signaling cascade regulates cell function and survival after stress stimulation. Equally robust studies reported dichotomous results suggesting both protective and detrimental effects of JNK during myocardial ischemia-reperfusion (I/R)," scientists in Lausanne, Switzerland report.
"The lack of a highly specific JNK inhibitor contributed to this controversy. We recently developed a cell-penetrating, protease-resistant peptide...
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Source: Blood Weekly (2007-05-17)
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