Cancer Vaccines
Eliminating Influence of Hyperactive Genes That Help Regulate Cell Development Shows Potential as Additional Target Therapy for Chronic Myelogenous Leukemia
Published in Blood Weekly, November 29th, 2007
When these genes, Rac1 and Rac2, become hyperactive, they prompt a protein linked to leukemia to stimulate the onset and spread of CML, a cancer that originates in blood marrow stem cells. But targeted inactivation of those genes, or using a molecule to nullify their hyperactivity, inhibits the onset of leukemia due to the 'Philadelphia chromosome' fusion protein called BCR-ABL, according to David...
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Source: Blood Weekly (2007-11-29)
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