Tissue Engineering

Study data from B.H. Kim and co-authors update knowledge of tissue engineering

Published in Blood Weekly, June 11th, 2009

"In this study, we examined the regulatory role of G-Rp1 on cell adhesion events mediated by beta 1-integrins (CD29). Using a U937 cell-cell adhesion assay, we found that exogenous G-Rp1 down-regulates CD29 activation in a dose-dependent manner, whereas G-Rg3 did not cause the same effect," scientists in South Korea report.

"However, G-Rp1 increased cell-fibronectin adhesion comparable to cytochalasin B, an actin cytoskeleton disruptor. Furthermore, G-Rp1 also blocked the rearrangement of actin at sites of cell-cell contact, indicating that the actin cytoskeleton may be a target of G-Rp1 action. Interestingly, G-Rp1 suppressed dephosphorylation of vasodilator-stimulated...

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Source: Blood Weekly (2009-06-11)

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