Microcirculation
Studies from University of Lubeck reveal new findings on microcirculation
Published in Blood Weekly, July 9th, 2009
"The activation of endothelial Ca2+-dependent K+-channels, K(Ca)3.1 (IKCa), and K(Ca)2.3 (SKCa) has been proposed to be a prerequisite for endothelial hyperpolarization, which subsequently hyperpolarizes and relaxes smooth muscle [endothelium-derived hyperpolarizing factor (EDHF)-type dilation] and initiates conducted dilations. Although EDHF is the main mediator of acetylcholine (ACh)-induced dilation in the murine skeletal microcirculation, the differential contribution of K(Ca)3.1 and K(Ca)2.3 is not known," researchers in Lubeck, Germany report.
"We assessed agonist-induced and conducted dilations as well as endothelial hyperpolarization in the cremaster...
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Source: Blood Weekly (2009-07-09)
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