Published in Blood Weekly, December 3rd, 2009
"This results in qualitative or quantitative abnormalities of the platelet receptor, alpha IIb-beta 3. The aim of this study was to identify the mutations in GT patients and to correlate these with patient phenotype. A total of 45 unrelated patients with GT were enrolled in the present study to identify the causative molecular defects, and also...
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Source: Blood Weekly (2009-12-03)
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