Published in Cancer Weekly, September 6th, 2011
"These alterations represent possible targets for investigational therapies that could act to specifically halt the aberrant growth of AML cells while limiting damage to normal cells. One such gene is the Fms-like tyrosine kinase 3 (FLT3) gene, which is mutated in approximately 30% of adult patients...
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Source: Cancer Weekly (2011-09-06)
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