Published in Cardiovascular Business Week, October 26th, 2004
Scientists in Canada report, "Heat shock (HS) proteins (Hsps) function in tissue protection through their chaperone activity and by interacting with cell signaling pathways to suppress apoptosis. [We] investigated the effect of HS treatment on the nuclear factor (NF)-kappaB signaling pathway in the angiotensin II (ANG II) model of inflammation."
Y. Chen and colleagues of Dalhousie University in Halifax described their study, "Male Sprague-Dawley rats were divided into sham and HS-, ANG II-, and HS + ANG II- treated groups. HS treatment was...
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