Published in Cardiovascular Business Week, November 7th, 2006
According to recent research from Australia, "Myocardial contractility is enhanced in transgenic (TG) mice with cardiac-restricted overexpression of the alpha1A -adrenergic receptors (alpha1A -AR). We tested the hypothesis that this enhanced inotropy protects against dysfunction and remodeling after myocardial infarction (MI)."
"We subjected alpha1A -TG and non-TG mice (NTG) to MI and determined changes in left ventricular (LV) function and diastolic dimension (LVDd) by...
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