Published in Cardiovascular Device Liability Week, May 4th, 2008
"In this study, we examined the effects of overexpression of a natural inhibitor of uPA, plasminogen activator inhibitor-1 (PAI-1), on the development of angiotensin (Ang) II-induced AAA in ApoE-deficient (ApoE (/) ) mice. Mice were treated with recombinant adenovirus containing either the human PAI-1 gene (Ad5. CMV. PAI-1) or the luciferase gene...
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