Published in Cardiovascular Week, June 12th, 2006
According to recent research from Australia, "This study evaluated the ability of A1 and A3 adenosine receptor (AR) agonism, and A1 ), A2A , A2B and A3 AR antagonism (revealing 'intrinsic' responses), to modify post-ischemic coronary dysfunction in mouse heart."
Griffith University-Southport investigators A.J. Zatta and colleagues wrote,...
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