Acute Myeloid Leukemia

Mutation may contribute to AML

Published in Clinical Oncology Week, August 25th, 2003

Researchers at Washington University School of Medicine in St. Louis have found that a chromosome defect often seen in human acute myeloid leukemia (AML) can cause the same disease in mice when combined with a genetic defect in a molecule known as a tyrosine kinase receptor.

The study is published online and in the August 5, 2003, issue of the Proceedings of the National Academy of Sciences USA.

"The findings indicate that tyrosine kinases may be an important class of cancer-causing genes in leukemia," said principal investigator Michael H. Tomasson, MD, assistant professor of genetics and of medicine. "It further suggests that drugs designed...

Want to see the full article?

Purchase this article for only $3.00

We're a pay-per-view site for premium content. If you'd like to purchase this article, it's only $3.00.

Welcome to NewsRx!

Learn more about a six-week, no-risk free trial of Clinical Oncology Week

Source: Clinical Oncology Week (2003-08-25)

NewsRx is Social

Top Medical Newsletters

Awards

Best Health/Healthcare Content, 2012
Best Health/Healthcare Content, 2011
Best e-Business Site, 2010
Best e-Business Site, 2009
Best e-Business Site, 2008
Best e-Business Site, 2007
Best e-Business Site, 2006
Best Healthcare Content, 2005
Best Overall Internet Site, 2005
Best Interactive Site, 2005

Facts & Stats

NewsRx also is available at LexisNexis, Gale, ProQuest, Factiva, Dialog, Thomson Reuters, NewsEdge, and Dow Jones.

Google 2010 PageRank: #2 Among Top Health News and Media Publications
Google 2010 PageRank: #2 Among Top Science Publications in Biology/Physiology
Google 2010 PageRank: #2 Among Top News and Media for the Business of Pharmaceuticals
Amazon's Alexa 2010 PageRank: #2 News and Media Site for the Pharmaceutical Industry