Aging

New aging study results reported from N. Kimura et al

Published in Disease Risk Factor Week, January 27th, 2008

"Impaired axonal transport may promote pathogenesis in neurodegenerative disorders, such as Alzheimer's disease (AD). We previously showed that tau, amyloid precursor protein (APP), and intracellular amyloid P-protein (A beta) accumulate in the nerve-ending fraction of aged monkey brains, perhaps because of impaired axonal transport," scientists writing in the Journal of Neuroscience Research report.

"In the present study, we assessed age-related changes of axonal transport motor proteins in aged monkey brains. Western blotting showed that kinesin, dynein, and dynactin (DYN) localizations dramatically changed with aging, and dynein level in nerve-ending fractions...

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Source: Disease Risk Factor Week (2008-01-27)

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