Parkinson Disease
Studies from L. Ma and co-researchers update current data on Parkinson disease
Published in Disease Risk Factor Week, February 28th, 2010
According to recent research published in the journal Neurochemical Research, "In Parkinson's disease substantia nigra neurons degenerate likely due to oxidative damage interacting with genetic risk factors. Here, SH-SY5Y cells expressing wild-type or A53T alpha-synuclein had increased sensitivity to methyl-4-phenylpyridinium iodide (MPP+), which induces mitochondrial dysfunction, and 6-hydroxydopamine (6-OHDA), which causes oxidative stress."
"Edaravone protected only against MPP+, and EGCG ((-)-epigallocatechin-3-O-gallate) protected only against 6-OHDA. Thus genomic responses to MPP+ and 6-OHDA in the presence of these antioxidants were analyzed using...
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