Published in Drug Law Weekly, November 14th, 2006
"The contribution of atypical PKC-zeta to ANG II-accelerated restenosis after endoluminal vascular injury was investigated by using the rat carotid balloon injury model," wrote J.H. Parmentier and colleagues, University of Tennessee.
"Exposure of injured arteries to ANG II resulted in an extensive neointimal thickening (1.9 times) compared with vehicle at day 14. Treatment with PKC-zeta antisense, but not scrambled, oligonucleotides reduced neointimal formation...
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