Insulin-Dependent Diabetes (Risk Factors)

Chemically-Induced Diabetes Resistance in Mice Linked to Elevated Antioxidant Levels

Published in Gene Therapy Weekly, August 23rd, 1999

Research at The Jackson Laboratory comparing mice bred for resistance or susceptibility to chemically-induced insulin-dependent (Type 1) diabetes suggests that the major difference is in the genetic control of antioxidant enzymes.

These enzymes help protect against pancreatic beta-cell destruction caused by free radical-mediated stress in both mice and humans.

The results add a new dimension in understanding why some humans with "high-risk" genes for developing Type 1 diabetes remain free of this disease. Identification of the gene or genes conferring such remarkable resistance could lead to therapies to boost natural defenses against diabetes and...

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Source: Gene Therapy Weekly (1999-08-23)

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