Published in Gene Therapy Weekly, August 23rd, 1999
These enzymes help protect against pancreatic beta-cell destruction caused by free radical-mediated stress in both mice and humans.
The results add a new dimension in understanding why some humans with "high-risk" genes for developing Type 1 diabetes remain free of this disease. Identification of the gene or genes conferring such remarkable resistance could lead to therapies to boost natural defenses against diabetes and...
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