Published in Gene Therapy Weekly, December 8th, 2005
"Somatostatin inhibits cell proliferation through interaction with its cellular receptor, SSTR. We have previously demonstrated that overexpression of individual SSTR-1 or SSTR-2 genes in receptor-negative pancreatic cancer cells inhibited cell proliferation."
"We hypothesize that reintroduction of SSTR genes back into pancreatic cancer cells might be an effective gene therapy strategy for pancreatic cancer,"...
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