Gene Therapy
Reports summarize gene therapy research from University of Texas, Health Science Center
Published in Gene Therapy Weekly, June 5th, 2008
New research, 'Smad4-dependent TGF-beta signaling suppresses RON receptor tyrosine kinase-dependent motility and invasion of pancreatic cancer cells,' is the subject of a report. According to recent research from the United States, "Transforming growth factorbeta (TGF-beta) signals through Smad-dependent and Smad-independent pathways. However, Smad signaling is altered by allelic deletion or intragenic mutation of the Smad4 gene in more than half of pancreatic ductal adenocarcinomas."
"We show here that loss of Smad4-dependent signaling leads to aberrant expression of RON, a phosphotyrosine kinase receptor, and that signaling by RON cooperates with Smad4-independent...
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Source: Gene Therapy Weekly (2008-06-05)
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