Angiotensins

Findings from Emory University in angiotensins reported

Published in Gene Therapy Weekly, June 12th, 2008

"Mitochondrial dysfunction is a prominent feature of most cardiovascular diseases. Angiotensin (Ang) II is an important stimulus for atherogenesis and hypertension; however, its effects on mitochondrial function remain unknown," scientists in the United States report.

"We hypothesized that Ang II could induce mitochondrial oxidative damage that in turn might decrease endothelial nitric oxide (NO center dot) bioavailability and promote vascular oxidative stress. The effect of Ang II on mitochondrial ROS, mitochondrial respiration, membrane potential, glutathione, and endothelial NO center dot was studied in isolated mitochondria and intact bovine aortic endothelial cells...

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Source: Gene Therapy Weekly (2008-06-12)

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