Enzyme Research
Scientists at University of Chicago describe research in enzyme research
Published in Gene Therapy Weekly, August 28th, 2008
Research findings, 'Phosphorylation of Bad at Thr-201 by JNK1 promotes glycolysis through activation of phosphofructokinase-1,' are discussed in a new report. "The mitogen-activated protein kinase JNK1 suppresses interleukin-3 withdrawal-induced cell death through phosphorylation of the BH3-only pro-apoptotic Bcl-2 family protein Bad at Thr-201. It is unknown whether JNK1 regulates glycolysis, an important metabolic process that is involved in cell survival, and if so, whether the regulation depends on Thr-201 phosphorylation of Bad," researchers in the United States report.
"Here we report that phosphorylation of Bad by JNK1 is required for glycolysis through...
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Source: Gene Therapy Weekly (2008-08-28)
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