Antisense Technology
Study findings from T. Ozaki et al broaden understanding of antisense technology
Published in Gene Therapy Weekly, February 11th, 2010
According to a study from Osaka, Japan, "In the inflamed liver, proinflammatory cytokines including TNF-alpha, IL-1 beta, and IFN-gamma stimulate the induction of iNOS gene expression, leading to excess production of NO and resulting in liver injury. The induction of iNOS is regulated by transactivation of the iNOS promoter with transcription factors such as nuclear factor kappa B and by posttranscriptional modifications such as mRNA stabilization."
"The synthetic glucocorticoid dexamethasone has been reported to inhibit iNOS induction, which may contribute to its inflammation-reducing effects. The objective was to investigate the mechanisms involved in the...
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Source: Gene Therapy Weekly (2010-02-11)
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