Neuropathic Pain
Data on neuropathic pain detailed by researchers at Kinki University, Division of Pharmacology
Published in Gene Therapy Weekly, September 2nd, 2010
Researchers detail in 'Upregulation of Ca(v)3.2 T-type calcium channels targeted by endogenous hydrogen sulfide contributes to maintenance of neuropathic pain,' new data in neuropathic pain. According to a study from Japan, "Hydrogen sulfide (H(2)S) formed from l-cysteine by multiple enzymes including cystathionine-gamma-lyase (CSE) is now considered a gasotransmitter in the mammalian body. Our previous studies have shown that H(2)S activates/sensitizes Ca(v)3.2 T-type Ca(2+) channels, leading to facilitation of somatic and visceral nociception, and that CSE-derived endogenous H(2)S participates in inflammatory pain."
"Here, we show novel evidence for involvement of the...
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Source: Gene Therapy Weekly (2010-09-02)
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