Life Sciences
Study results from University Hospital, Division of Clinical Immunology update understanding of life sciences
Published in Genetics and Environmental Law Weekly, September 12th, 2009
Research findings, 'Clock gene modulation by TNF-alpha depends on calcium and p38 MAP kinase signaling,' are discussed in a new report. According to a study from Zurich, Switzerland, "A 24-h treatment with the cytokine tumor necrosis factor-alpha (TNF-alpha) suppresses transcription of E-box-driven clock genes (D-site albumin promoter binding protein, Dbp; Tyrotroph embryonic factor, Tef ; Hepatic leukemia factor, Hlf; Period homolog to Drosophila 1/2/3, Per1, Per2, and Per3) by yet unknown molecular mechanisms. The attenuation of clock genes has been suggested as a putative cause for the development of sickness behavior syndrome in infectious and autoimmune diseases."
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