Published in Health Business Week, November 12th, 2004
"Gap junctions are thought to be necessary for proper tissue function. However, no clear hepatic phenotype has been described in patients lacking connexin 32 (Cx32), the principal gap junction in liver," researchers in the United States explained.
"To determine the physiological role of Cx32 in liver," P.R.A.V. Correa and colleagues at Yale University "compared the response of wild type and Cx32-deficient mice to endotoxin, since this stress increases serum levels of hormones that bind to receptors that are asymmetrically distributed across the hepatic lobule."
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