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Ataxia Telangiectasia
Protein 'chatter' linked to cancer activation
August 27th, 2007
Scientists have found the existence of cross-talk between human chromosome ends and the protein complexes central to the stability of the entire human genome, a “chat” that contributes to cancer development. The research, published today in Nature Structural & Molecular Biology, sheds new light on the pathology of three related but non-curable cancer-predisposed human disorders: ataxia telangiectasia, ataxia telangiectasia-like disorder, and Nijmegen breakage syndrome. “The proteins whose deficiency is responsible for these three human disorders have the job of ensuring that chromosome ends are maintained and protected,” says Xu-Dong Zhu, assistant...
Source: Health & Medicine Week (2007-08-27)
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