Hematology
HO-2 gene deletion may protect striatal cells from Hb oxidative toxicity
Published in Hematology Week, December 19th, 2005
According to recent research published in the Journal of Cerebral Blood Flow and Metabolism, "The heme oxygenase (HO) enzymes catalyze the rate-limiting step in the breakdown of heme to iron, carbon monoxide, and biliverdin. A prior cell culture study demonstrated that deletion of HO-2, the isoform constitutively expressed in neurons, attenuated hemoglobin (Hb) neurotoxicity."
Y. Qu and colleagues at Thomas Jefferson University in Philadelphia explained, "The present study tested the hypothesis that HO-2 gene deletion is...
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Source: Hematology Week (2005-12-19)
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