Cardioprotection

Researchers at Medical College of Georgia, Medical College have published new data on cardioprotection

Published in Hematology Week, June 2nd, 2008

According to recent research from the United States, "The mitochondrial permeability transition (MPT) pore may serve as the end-effector of cardio protective mechanisms, namely the mitochondrial K-ATP channels and glycogen synthase kinase-3 beta (GSK-3 beta). We recently showed that augmented MPT pore induction contributes to pressure overload-induced exacerbation of infarct size."

"This study tests the hypotheses that (i) elevation in perfusion pressure attenuates cardioprotection associated with activation of mitochondrial K-ATP channels or inhibition of GSK-3 beta and (ii) perfusion pressure modulates the regulation of the MPT pore by mitochondrial K-ATP channels...

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Source: Hematology Week (2008-06-02)

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