Pathogenesis

Th1 Protective in Hepatitis-Related Fulminant Disease

Published in Hepatitis Weekly, June 24th, 1996

A study demonstrating a protective role for Th1 cells, but not for Th2, in the pathogenesis of murine hepatitis virus-3-induced liver failure could enhance the understanding of hepatitis-related fulminant disease in humans.

Fulminant viral hepatitis remains a severe disease, with a mortality rate approaching 80 percent. In earlier studies researcher Marc Pope and colleagues from Canada's University of Toronto used a murine model of fulminant viral hepatitis caused by murine hepatitis virus strain 3 (MHV-3) to study the pathogenesis of liver necrosis. MHV-3 is a single-stranded, positive-sense RNA virus belonging to the Coronaviridae family, which produces a...

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Source: Hepatitis Weekly (1996-06-24)

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