Published in Hepatitis Weekly, December 23rd, 2002
Noting that "the mechanism by which HCC cells resist apoptosis induced by various treatment modalities is poorly understood," they "assessed constitutive and inducible activation of NF-kappaB in hepatitis B virus (HBV) positive (Hep3B) and in hepatitis virus negative (Chang, HepG2) HCC, cells, as well as the role of known inhibitors of NF-kappaB activity."
P.J. Chiao and colleagues listed their study findings:
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