Research findings, 'Aetiopathogenesis of autoimmune hepatitis,' are discussed in a new report. "Interface hepatitis, histological hallmark of autoimmune hepatitis (AIH), is a dense portal mononuclear cell infiltrate invading the parenchyma, consisting of CD4 and CD8 T-lymphocytes, monocytes/macrophages and plasma cells. What triggers AIH is unknown, but there is evidence that failure of immunehomeostasis plays a permissive role allowing liver autoantigen-specific cells to attack hepatocytes," scientists in London, the United Kingdom report.
"Damage is likely to be orchestrated by CD4 T-lymphocytes recognizing an autoantigenic liver peptide. For autoimmunity to arise,...
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