Life Sciences
Researchers at Stanford University target life sciences
Published in Immunotherapy Weekly, July 1st, 2009
Researchers detail in 'Loss of the desmosomal protein perp enhances the phenotypic effects of pemphigus vulgaris autoantibodies,' new data in life sciences. According to recent research published in the Journal of Investigative Dermatology, "Pemphigus vulgaris (PV) is an autoimmune bullous disease in which autoantibodies against proteins of the desmosomal adhesion complex perturb desmosomal function, leading to intercellular adhesion defects in the oral mucosa and skin. Previous studies have demonstrated a central role for downregulation of the desmosomal cadherin desmoglein 3 (DSG3) in the pathogenesis of PV."
"However, the effects of non-cadherin desmosomal...
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Source: Immunotherapy Weekly (2009-07-01)
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