Boston University Medical Center
Gene expression in severely emphysematous lung tissue characterized
Published in Medical Device Law Weekly, March 6th, 2005
"The mechanism by which inhaled smoke causes the anatomic lesions and physiologic impairment of chronic obstructive pulmonary disease remains unknown," scientists in the United States explained.
In a recent study, A. Spira and coauthors at the Boston University Medical Center "used high-density microarrays to measure gene expression in severely emphysematous lung tissue removed from smokers at lung volume reduction surgery (LVRS), and normal or mildly emphysematous lung tissue from smokers undergoing resection of pulmonary nodules."
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