Plasmodium chabaudi (Malaria)

Gene-Controlled Resistance Subject to Superior Hormonal Imprinting

Published in Medical Letter on the CDC and FDA, July 14th, 1997

Testosterone administered to female mice affected their resistance to Plasmodium chabaudi malaria, even after normal hormone levels were restored.

Testosterone induces susceptibility to P. chabaudi malaria by imposing restrictions on the mechanisms that mediate resistance, said W.P.M. Benten and colleagues, Heinrich Heine University, Dusseldorf, Germany.

Benten et al. performed a study to determine whether these restrictions cease after testosterone is withdrawn ("Testosterone-Induced Susceptibility to Plasmodium chabaudi Malaria: Persistence after Withdrawal of Testosterone," Journal of Endocrinology, 1997;153:275-281).

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Source: Medical Letter on the CDC and FDA (1997-07-14)

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