Cardiovascular Disease
Inhibition of rapamycin target modulates adhesion molecule expression
Published in Medical Letter on the CDC and FDA, October 8th, 2006
According to a report from the United States, "Neointimal hyperplasia often follows angioplasty-induced arterial injury or stenting and results in restenosis. Previous reports have suggested that arterial injury activates complement, which amplifies inflammatory responses that may initiate and sustain neointimal hyperplasia. The effects of rapamycin on complement-induced expression of intracellular adhesion molecules (ICAMs) were examined in porcine arterial endothelial cell (PAEC) line that was transformed with large T antigen."
"Porcine...
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