Published in Mental Health Business Week, September 30th, 2006
"There is ample evidence for the involvement of oxidative stress in mitochondrial DNA damage and repair mechanisms in PD. The human MutY homolog (hMUTYH) which removes misincorporated adenine opposite 8-oxoG in DNA functions in post-replication, and is localized in the nuclei and mitochondria.
"We hypothesized that hMUTYH is involved in the disease process of PD. To test our hypothesis, we performed immunohistochemical and biochemical studies on brains of patients with PD and those of control...
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