Published in Obesity and Diabetes Week, January 2nd, 2006
According to recent research from Turkey, "The defects identified in the mechanical activity of the hearts from type 1 diabetic animals include alteration of Ca2+ signaling via changes in critical processes that regulate intracellular Ca2+ concentration. These defects result partially from a dysfunction of cardiac ryanodine receptor calcium release channel (RyR2)."
N. Yaras and colleagues at Ankara University designed the current investigation "to determine whether the properties of the...
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