Atherosclerosis
Fatty acids cause alterations of hASMC proteoglycans that increase affinity for LDL
Published in Obesity and Diabetes Week, February 20th, 2006
According to a study from Sweden, "The dyslipidemia of insulin resistance, with high levels of albumin-bound fatty acids, is a strong cardiovascular disease risk. Human arterial smooth muscle cell (hASMC) matrix proteoglycans (PGs) contribute to the retention of apoB lipoproteins in the intima, a possible key step in atherogenesis."
M. Rodriguez-Lee and colleagues working with Gothenburg University assessed "the effects of high NEFA levels on the PGs secreted by hASMCs and whether these effects might alter the PG...
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