Epilepsy

Researchers at Baylor College of Medicine, College of Medicine target epilepsy

Published in Pain and Central Nervous System Week, April 13th, 2009

According to a study from the United States, "Absence seizures are a leading form of childhood epilepsy. Human and mouse P/Q-type calcium channel gene mutations initiate a complex absence epilepsy and ataxia phenotype, and in mice, secondarily elevate neuronal low-voltage-activated T-type calcium currents."

"These currents influence thalamocortical network activity and contribute to the generation of cortical spike-wave discharges (SWDs) associated with absence seizures. To address whether enhanced thalamocortical T-type currents suffice to induce an epileptic phenotype, two BAC transgenic mouse lines overexpressing the Cacna1g gene for alpha 1GT-type calcium channels...

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Source: Pain and Central Nervous System Week (2009-04-13)

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