Published in Pharma Investments, Ventures and Law Weekly, September 19th, 2004
According to published research from France, "the dopamine transporter (DAT) plays a critical role in calibrating the duration and intensity of dopamine (DA) neurotransmission. Mice in which the DAT gene has been genetically deleted exhibit constitutively high levels of extrasynaptic DA and spontaneous hyperactivity. Numerous studies have characterized the adaptive molecular, physiological, and behavioral consequences of abnormal DA neurotransmission in these mice."
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