Medical University of South Carolina, Charleston
Human eNOS gene delivery renders aid to injured hearts
Published in Physician Law Weekly, May 25th, 2005
"Nitric oxide (NO) has been shown to play a key role in the regulation of cardiac hypertrophy and fibrosis in response to myocardial ischemia in part by antagonizing the action of angiotensin II (Ang II). In this study, we investigated the potential protective role of human endothelial nitric oxide synthase (eNOS) in left ventricular (LV) remodeling after myocardial infarction (MI) by a somatic gene transfer approach," scientists in the United States report.
"Male Wistar rats underwent coronary artery ligation to induce MI 1 week after surgery, adenovirus encoding the human eNOS or luciferase...
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Source: Physician Law Weekly (2005-05-25)
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