Published in Proteomics Weekly, December 20th, 2004
"Signaling mediated by the cellular kinase mammalian target of rapamycin (mTOR) activates cap-dependent translation under normal (nonstressed) conditions," wrote S.B. Kudchodkar and colleagues, University of Pennsylvania.
"However," the authors explained, "translation is inhibited by cellular stress responses or rapamycin treatment, which inhibit mTOR kinase activity."
"We show that during human cytomegalovirus (HCMV)...
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